Jake Ormond

Jake received his M.Sc. studying the molecular pathways underlying synaptic plasticity in the marine mollusc Aplysia with Dr. Wayne Sossin at the Montreal Neurological Institute, McGill University. He then went on to complete a Ph.D. with Dr. Melanie Woodin at the University of Toronto, using in vitro electrophysiological methods to investigate the role of inhibitory plasticity in hippocampal long-term potentiation. Currently, Jake is examining the differential contributions made by grid cells at various locations along the dorsal to ventral extent of the medial entorhinal cortex to hippocampal place field expression.

Recent Publications

  1. Disinhibition mediates a form of hippocampal long-term potentiation in area CA1.
    Ormond J, Woodin MA.
    PLoS One. 2009 Sep 29;4(9):e7224.
  2. Mechanisms regulating ApTrkl, a Trk-like receptor in Aplysia sensory neurons.
    Nagakura I, Ormond J, Sossin WS.
    J Neurosci Res. 2008 Oct;86(13):2876-83.
  3. ApTrkl, a Trk-like receptor, mediates serotonin- dependent ERK activation and long-term facilitation in Aplysia sensory neurons.
    Ormond J, Hislop J, Zhao Y, Webb N, Vaillaincourt F, Dyer JR, Ferraro G, Barker P, Martin KC, Sossin WS.
    Neuron. 2004 Nov 18;44(4):715-28. Paper.